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Acute adriamycin-induced cardiotoxicity is exacerbated by angiotension II
Taskin, Eylem; Kindap, Elvan Kunduz; Ozdogan, Kalender; Aycan, Mukerrem Betul Yerer; Dursun, Nurcan (SPRINGER, 2016)Adriamycin (ADR) increases the production of reactive oxygen species (ROS), which diminishes mitochondrial function. Angiotensin-II stimulates mitochondrial ROS generation. The aim of the study was to examine whether ... -
The cooperative effect of local angiotensin-II in liver with adriamycin hepatotoxicity on mitochondria.
Taşkın, Eylem; Güven, Celal; Şahin, Leyla; Dursun, Nurcan (Medical Science International Publishing, 2016)Background: Adriamycin (ADR) is a drug used clinically for anticancer treatment; however, it causes adverse effects in the liver. The mechanism by which these adverse effects occur remains unclear, impeding efforts to ... -
Inhibition of Angiotensin-II Production Increases Susceptibility to Acute Ischemia/Reperfusion Arrhythmia
Taskin, Eylem; Tuncer, Kadir Ali; Guven, Celal; Kaya, Salih Tunc; Dursun, Nurcan (INT SCIENTIFIC LITERATURE, INC, 2016)Background: Myocardial ischemia and reperfusion lead to impairment of electrolyte balance and, eventually, lethal arrhythmias. The aim of this study was to investigate the effects of pharmacological inhibition of angiotensin-II ... -
The restoration of kidney mitochondria function by inhibition of angiotensin-II production in rats with acute adriamycin-induced nephrotoxicity
Taşkın, Eylem; Özdoğan, Kalender; Kindap, Elvan Kunduz; Dursun, Nurcan (Informahealthcare, 2014)Adriamycin (ADR) is commonly used for many solid tumor treatments. Its clinical utility is, however, largely limited by the adverse reactions, are known to be nephrotoxic. The mechanism by which it induces kidney damage ... -
The restoration of kidney mitochondria function by inhibition of angiotensin-II production in rats with acute adriamycin-induced nephrotoxicity
Taskin, Eylem; Ozdogan, Kalender; Kindap, Elvan Kunduz; Dursun, Nurcan (TAYLOR & FRANCIS LTD, 2014)Adriamycin (ADR) is commonly used for many solid tumor treatments. Its clinical utility is, however, largely limited by the adverse reactions, are known to be nephrotoxic. The mechanism by which it induces kidney damage ...
