Advanced Search

Show simple item record

dc.contributor.authorAkman, Özlem
dc.contributor.authorKarson, A
dc.contributor.authorAtes, N
dc.contributor.authorOnat, F
dc.contributor.editorİstanbul Bilim Üniversitesi, Tıp Fakültesi.en_US
dc.date.accessioned2014-08-05T14:42:17Z
dc.date.available2014-08-05T14:42:17Z
dc.date.issued2010
dc.identifier.citationAkman O, Karson A, Ates N, Onat F. Perirhinal Cortical Kindling In Genetic Absence Epilepsy Rat Strains. Epilepsia. 2010; 51(Suppl 4):1–189. 9th European Congress on Epileptology, Rhodes, Greece, 27 June–1 July 2010 doi: 10.1111/j.1528-1167.2010.02658.xen_US
dc.identifier.issn1528-1167
dc.identifier.urihttps://hdl.handle.net/11446/404en_US
dc.description.abstractPurpose: The genetic models of absence epilepsy, GAERS and WAG/ Rij rat strains, are resistant to progression of partial seizures induced by amygdaloid and hippocampal kindling. Perirhinal cortex is a crucial area for the secondary generalization of partial seizures. Kindling epileptogeniticy is propagated from perirhinal cortex to other areas that are responsible for generalized seizures. Therefore we focused on perirhinal cortical kindling in both epileptic rat strains and compared them with Wistar rats.en_US
dc.description.urihttp://onlinelibrary.wiley.com/doi/10.1111/j.1528-1167.2010.02658.x/pdfen_US
dc.language.isoengen_US
dc.publisherWiley Online Libraryen_US
dc.rightsinfo:eu-repo/semantics/openAccessen_US
dc.titlePerirhinal cortical kindling in genetic absence epilepsy rat strainsen_US
dc.typeconferenceObjecten_US
dc.relation.journalEpilepsiaen_US
dc.contributor.departmentDBÜen_US
dc.identifier.volume51
dc.identifier.startpage18
dc.identifier.endpage18
dc.contributor.authorIDTR45271en_US
dc.contributor.authorIDTR155950en_US
dc.contributor.authorIDTR25866en_US
dc.contributor.authorIDTR122028en_US
dc.relation.publicationcategoryBelirsizen_US


Files in this item

Thumbnail

This item appears in the following Collection(s)

Show simple item record