dc.contributor.author | Güdücü, Nilgün | |
dc.contributor.author | Görmüş, Uzay | |
dc.contributor.author | Alp, E | |
dc.contributor.author | Kavak, Zehra Neşe | |
dc.contributor.author | Dünder, İlkkan | |
dc.date.accessioned | 2014-11-10T12:48:22Z | |
dc.date.available | 2014-11-10T12:48:22Z | |
dc.date.issued | 2014 | |
dc.identifier.citation | Guducu N, Gormus U, Alp E, Kavak ZN, Dunder I. Reciprocal action of pentraxin-3 and Crp in women with polycystic ovary syndrome. European Journal of Inflammation. 2014; 12(1): 161-166. | en_US |
dc.identifier.issn | 1721-727X | |
dc.identifier.uri | www.biolifesas.org | en_US |
dc.identifier.uri | https://hdl.handle.net/11446/518 | en_US |
dc.description | İstanbul Bilim Üniversitesi, Tıp Fakültesi. | en_US |
dc.description.abstract | The aim of this study was to investigate the relationship between pentraxin-3 and other biochemical
parameters in women with polycystıc ovary syndrome (PCOS). We compared 58 women with PCOS to
34 body mass index- and age-matched normally menstruating healthy controls. Women with PCOS had
significantly higher DHEA-S, free testosterone, LH and FAI, but lower pentraxin-3 levels when compared
to healthy controls (0.86±0.21 and 0.91±0.14 respectively, p=0.014). Levels of CRP and lipoprotein-a
were higher in the PCOS group. Overweight PCOS had significantly higher insulin, HOMA-IR, FAI,
free testosterone and CRP and statistically significantly lower HDL and SHBG levels when compared to
controls. Pentraxin-3 levels of obese and normal weight PCOS were similar. CRP and pentraxin-3 might
contribute reciprocally to metabolic events and chronic low-grade inflammation in women with PCOS. | en_US |
dc.language.iso | eng | en_US |
dc.publisher | Biolifesas.org | en_US |
dc.rights | info:eu-repo/semantics/embargoedAccess | en_US |
dc.title | Reciprocal action of pentraxin-3 and Crp in women with polycystic ovary syndrome | en_US |
dc.type | article | en_US |
dc.relation.journal | European Journal of Inflammation | en_US |
dc.department | DBÜ, Tıp Fakültesi | en_US |
dc.identifier.issue | 1 | |
dc.identifier.volume | 12 | |
dc.identifier.startpage | 161 | |
dc.identifier.endpage | 166 | |
dc.contributor.authorID | TR123126 | en_US |
dc.contributor.authorID | TR34467 | en_US |
dc.contributor.authorID | TR173362 | en_US |
dc.contributor.authorID | TR26039 | en_US |
dc.relation.publicationcategory | Belirsiz | en_US |