Analysis of the complement pathway mutations in atypical hemolytic uremic syndrome (AHUS)
Künye
Hancer VS, Guvenc S, Hindilerden F, Buyukdogan M, Diz-Kucukkaya R. (2014). Analysis of the complement pathway mutations in atypical hemolytic uremic syndrome (AHUS). Thrombosis Research. 2014: S35–S123. 23rd MLTD Biennial International Congress on Thrombosis. Valencia, May 14-17, 2014Özet
Background: HUS is characterized by hemolytic anemia,
thrombocytopenia, and renal failure caused by platelet thrombi in
the microcirculation of the kidney and other organs. Typical HUS is
triggered by infectious agents such as strains of E. coli that produce
powerful Shiga-like exotoxins, whereas AHUS can be genetic,
acquired, or idiopathic.
Kaynak
Thrombosis ResearchCilt
133Sayı
S3Bağlantı
https://hdl.handle.net/11446/676https://www.clinicalkey.com/#!/content/journal/1-s2.0-S0049384814502642
